Secondary immunodeficiency

Secondary immunodeficiency - wani take hakkin da tsarin na rigakafi da cewa tasowa a cikin postnatal lokaci. Yana kuma iya faruwa a manya.

Akwai iri uku sakandare immunodeficiencies: jawo, tsiwirwirinsu, maras wata-wata.

Daya sananne misali ne ya samu wani nau'i na cutar kanjamau. Sosai rigakafi, a wannan yanayin, saboda da sakamako na mutum immunodeficiency cutar.

Jawo nau'i na faruwa a ƙarƙashin rinjayar takamaiman haddasawa, wanda kai ta zuwa ga abin da ya faru. Wadannan sun hada da X-ray radiation, corticosteroids, cytostatic far, tiyata, rauni, illa rigakafi, wanda tasowa na biyu to tamkar cuta (koda, hanta da cutar, da ciwon sukari, ciwon daji).

Idan akwai wani kwakkwaran dalili ba cewa yana sa take hakkin reactivity sakandare immunodeficiency aka kira maras wata-wata. Asibiti shi ne bayyana akai-akai maimaita cututtuka na kumburi halitta a cikin broncho-na huhu na'ura, paranasal sinuses, urogenital, gastrointestinal fili, da idanu, fata, taushi da kyallen takarda, wadda aka sa ta opportunistic pathogens. Saboda haka, lingering, na kullum, sau da yawa relapsing kumburi tafiyar matakai na daban-daban sarrafawa a manya sanin da na asibiti manifestations na sakandare immunodeficiency jihohi.

The rinjaye nau'i na jihar data ne maras wata-wata. Primary immunodeficiency halin da sakandare a cewa shi dogara ne a kan m rigakafi da tsarin cuta.

Lokacin da cutar da ake gaji da damuwa da aiki na duk tsarin raka'a: T, B cell, phagocytic, gaba. Don gane marasa lafiya da irin wannan lahani ya kasu kashi uku kungiyoyin:

- ya bayyana kin amincewa da rigakafi, wanda ake hada tare da canji na sigogi.

- akwai kawai ãyõyin rigakafi rashi da cewa ba tare da wani canji na sigogi.

- Akwai canje-canje a cikin sigogi da cewa ba tare da ãyõyin rigakafi rashi.

Secondary immunodeficiency aka gano ta hanyar dakin gwaje-gwaje gwaje-gwaje: kafiya na rigakafi da matsayi (yawan leukocytes aka bincika, subpopulations na B da kuma T lymphocytes, matakan immunoglobulin M, G, A, phagocytosis). Ƙarin hanyoyin hada da katin shaida na comorbidities kuma ta kara magani. Kamar yadda ƙaddara da ba-takamaiman Manuniya na m lokaci, interferon matsayi. Bincikowa da wani muhimmanci bincike ainihin wannan factor cewa gano wannan jiha. Kawai bayan duk bincike gwaje-gwaje da aka sanya a takamaiman magani far, wanda ke taimakawa wajen hana ci gaban irin wannan cututtuka kamar sakandare immunodeficiency.

A lura da wannan yanayin da aka yi a daidai da da nauyi da kuma comorbidities.

A raunuka Kwayoyin na zuwa monocyte-macrophage hadaddun amfani polioksidony, likopid, molgramostim, filgramostim, Buffy gashi.

Lahani a salon salula rigakafi bukatar polioksidonija, taktivina, tamopina, imunofana, timogena, timolina, myelopid.

Canzawa far bada shawarar a cuta da humoral. Aiwatar Sandoglobulin, Octagam, Intraglobin, immunoglobulin biaven, pentaglobin.

Secondary immunodeficiency a lokacin asibiti magani warke bayan 20-30 kwanaki. A nan gaba, marasa lafiya ya kamata a gani kafin likita-immunologist da kuma kwararru na manyan cututtuka. A lokacin exacerbation da cutar bukatar sake magani, ga abin da na sama formulations aka yi amfani.