Polycythemia gaskiya. Haddasawa, Alamun, ganewar asali, Jiyya

Polycythemia - shi ne wata cuta wadda za a iya ƙaddara da neman kawai a mutum ta fuskar. Kuma idan muka gudanar da wani bincike Nazarin, da shakka, kuma ba ya zama. A likita wallafe-wallafen akwai wasu sunaye da wannan Pathology: erythremia, Vakeza cuta. Ko da kuwa da lokaci, cutar babban mai babbar barazana ga rayuwar dan Adam. A wannan labarin, za mu tattauna a zurfafe ginshikai na da ya faru, da farko bayyanar cututtuka, saukarwa da jiyya miƙa.

janar bayanai

Karkashin polycythemia tana nufin myeloproliferative jini da ciwon daji a wadda ta bargo samar ja jini Kwayoyin a matsanancin yawa. Don a karami har wasu enzyme aka gyara karuwa ne a lura, wato leucocytes da platelets.

Red jini Kwayoyin (RBCs in ba haka ba) tare da cikakken oxygen, duk mutum Kwayoyin, haihuwa da shi daga cikin huhu zuwa tsarin na ciki gabobin. Su ma alhakin kau da carbon dioxide daga kyallen takarda da kuma safarar shi zuwa ga huhu ga m exhalation.

Erythrocytes ake samar ci gaba a cikin bargo. Yana da wani spongiform jam'i kyallen takarda sarrafa a cikin kashi, kuma shi ne ke da alhakin aiwatar da hematopoiesis.

Leukocytes - farin jini Kwayoyin cewa taimake yaki cututtuka. Platelets ne anuclear cell, niƙaƙƙun gaɓãɓuwa da tashin hankali jijiyoyin bugun gini mutunci. Bã su da ikon bi da juna da kuma occlude bude, sa'an nan tsayawa da zub da jini.

Polycythemia gaskiya ne halin da ya wuce kima samar da ja jini Kwayoyin.

Yaduwar

Wannan Pathology ana bincikar lafiya a manya, amma zai iya faruwa a matasa da kuma yara. Da cutar ba za a iya ji na dogon lokaci, da cewa shi ne asymptomatic. A cewar nazarin, da talakawan shekaru na marasa lafiya jeri daga 60 zuwa 79 game da shekarun. Sun mutane suna da yawa m iya samun rashin lafiya, amma da cutar gudanar su da yawa wuya. Da wakilan da karfi jima'i, idan ka yi imani da statistics, sau da yawa more akai-akai kamu da polycythemia.

pathogenesis

Mafi yawa daga cikin matsalar lafiya hade da cuta, ya auku saboda da m karuwa a yawan ja jini Kwayoyin. A sakamakon haka, da jini zama ɓarna, lokacin farin ciki.

A daya hannun, ta ƙara ta danko tsokani da samuwar clots (thrombi). Suna iya tsoma baki tare da al'ada jini ya kwarara ta cikin jijiyoyi da kuma jijiyoyinmu. Irin wannan halin da ake ciki shi ne sau da yawa a cikin hanyar shanyewar jiki da kuma zuciya harin. The abu ne cewa, m jini a sau da yawa hankali tasoshin. The zuciya yana zuwa exert mafi kokarin zahiri tura ta.

Inda hakan na jini ya kwarara ba da damar kayan ciki kafin su sami ake bukata adadin oxygen. Wannan entails ci gaban da zuciya rashin cin nasara, ciwon kai, angina, wani rauni da sauran kiwon lafiya da matsaloli, wanda bai kamata a bar unattended.

rarrabuwa na cututtuka

• I. A farko.

1. Yana yana for 5 shekaru ko fiye.
2. The baƙin ciki na al'ada size.
3. Jini gwaje-gwaje nuna matsakaiciya karuwa a yawan ja jini Kwayoyin.
4. Matsalolin da wuya kamu.

• II A. polycythemic mataki.

1. Duration daga 5 zuwa 15 game da shekarun.
2. Akwai wata karuwa da wasu gabobin (baƙin ciki, hanta), zub da jini da kuma thrombosis.
3. Yankunan da ƙari a cikin baƙin ciki da kanta ba.
4. Zub da jini zai iya tsokana baƙin ƙarfe karanci a cikin jiki.
5. A cikin bincike na jini akwai wani m karuwa a ja da maikacin jini, da farin jini Kwayoyin da platelets.

• II mataki B. polycythemic da myeloid metaplasia baƙin ciki.

1. Na nazarin nuna wani ƙara abun ciki na duk da maikacin jini, fãce lymphocytes.
2. Akwai neoplastic aiwatar a cikin baƙin ciki.
3. The asibiti hoto na ãr bayyana, thrombosis, zub da jini.
4. A bargo ne a hankali a hankali samuwar scars.

• III. M mataki.

1. A cikin jini, akwai mai kaifi karu a erythrocytes, platelets da leukocytes.
2. Akwai pronounced karuwa a cikin size na baƙin ciki, kuma hanta.
3. Wannan lokaci yawanci tasowa a cikin shekaru 20 bayan tabbatar da ganewar asali.
4. A cutar za a iya canza kama zuwa wani m ko na kullum cutar sankarar bargo.

A Sanadin cutar

Abin baƙin ciki, yanzu masana ba zai iya ce abin da dalilai kai ga ci gaban wannan cututtuka kamar polycythemia gaskiya.

Mai karkata zuwa kwayar-kayyade ka'idar. A cewar ta, musamman ƙwayoyin cuta (akwai game da 15 guda) ana gabatar a cikin jikin mutum da kuma a karkashin tasirin wasu dalilai da cewa adversely shafi rigakafi defenses, shiga cikin bargo da kuma Lymph nodes. A Kwayoyin sun sa'an nan maimakon a ripening hanzari fara raba ku kuma riɓaɓɓanya, kafa sabuwar niƙaƙƙun.

A daya hannun, polycythemia dalilin iya lurking a hereditary predisposition. Masana kimiyya sun tabbatar da cewa wannan rashin lafiya ne mafi yiwuwa don rufe zumunta na rashin lafiya mutum, da kuma mutane tare da mai illa tsarin chromosomes.

Predisposing zuwa cuta dalilai

• X-ray sakawa a iska mai guba, ionizing radiation.
• Hanji cututtuka.
• Ƙwayoyin cuta.
• Da tarin fuka.
• Surgery.
• M danniya.
• Dogon lokacin da yin amfani da wasu kungiyoyin na kwayoyi.

asibiti hoto

An fara daga mataki na biyu na cuta, a cikin pathological aiwatar ake kõma kusan duk tsarin da kayan ciki. Aka jera a kasa ne na ra'ayin wani ji daga mãsu haƙuri.

• Wani rauni da wani haunting ji na gajiya.
• Karuwan sweating.
• A ban mamaki dakushe yi.
• Mai tsananin ciwon kai.
• memory hanawa.

Polycythemia gaskiya iya a tare da wadannan cututtuka. A kowane hali su mai tsanani dabam.

• Jijiyoyinmu da kuma canja ya dafa da fata. Marasa lafiya lura gaban fayyace jijiyoyinmu. A wannan cuta fata halin da m-tinged ceri, shi ne musamman m a kan fallasa sassa na jiki (harshe, hannuwa, fuska). Lebe ne blue, idanunsa da jũna bloodshot. Wannan canji a cikin bayyanar ne saboda da ambaliya da surface jini, da kuma wani alama slowdown a gabatarwa.
• Itchy fata. Wannan alama ya auku a 40% na lokuta.
• Rodonalgia (dõgẽwa kona zafi a dubaru na yatsunsu da yatsun kafa, wanda suna tare da redness na fata). Ana zargin wannan alama da ake dangantawa da ya karu da matakan jini platelets da kuma samuwar mikrotrombov.
• Kara girman da baƙin ciki.
• A bayyanar ulcers a ciki. Saboda kananan jirgin ruwa thrombosis mucosa jiki hasarar da juriya ga Helicobacter pylori.
• Jini clots. Su haddasawa an bayyana ta da karin jini danko da kuma canje-canje a cikin jijiyoyin bugun gini katangu. Kamar yadda mai mulkin, irin wannan halin da ake ciki take kaiwa zuwa rushewa daga jini wurare dabam dabam a cikin ƙananan wata gabar jiki, cerebral da jijiyoyin zuciya tasoshin.
• Pain a kafafu.
• Tã zub da jini daga gumis.

bincikowa da

Da farko likita tattara da cikakken likita tarihi. Ya iya tambaya mai yawan ayoyi tambayoyi: a lõkacin da ta bayyana rashin jin daɗi / breathlessness / m rashin jin daɗi, da dai sauransu. Shi ne daidai da muhimmanci domin sanin gaban kullum cututtuka, miyagun halaye, zai yiwu lamba tare da mai guba abubuwa ...

Sa'an nan, wani jiki jarrabawa. A gwani kayyade launi fata. By palpation da tapping bayyana wani kara girman baƙin ciki ko hanta.

Domin tabbatar da cuta mai m jini gwaje-gwaje ake nada. Idan haƙuri yana da wannan Pathology, wannan sakamakon su ne kamar haka:

• Kara yawan ja jini Kwayoyin.
• Dagagge sigogi hematocrit (kashi na ja da maikacin jini).
• High haemoglobin sigogi.
• Low matakan erythropoietin. Wannan hormone ne ke da alhakin ruri daga cikin bargo a samar da sabon ja jini Kwayoyin.

Ganewar asali ma yakan haifar da wani kwakwalwa biopsy da aspirate. A farko version na binciken ya shafi ci daga cikin ruwa bangaren kwakwalwa, da kuma biopsy - m bijiro da batun kafa.

polycythemia cuta tabbatar da gwaje-gwaje domin gene maye gurbi.

Abin da ya kamata a bi?

Gaba daya shawo kan cutar kamar polycythemia gaskiya, shi ne ba zai yiwu. Wannan shi ne dalilin da ya sa far aka mayar da hankali musamman kan rage asibiti manifestations da rage thrombotic rikitarwa.

Marasa lafiya da aka farko sanya bloodletting. Wannan hanya yakan haifar da wani kau na karamin adadin jini (daga 200 zuwa 400 game da ml) da curative niyyar. Wajibi ne ga daidaita da yawa sigogi na jini da kuma rage danko.

Marasa lafiya yawanci wajabta "asfirin" domin rage hadarin tasowa daban-daban iri thrombotic rikitarwa.

Jiyyar cutar sankara da ake amfani da su kula da al'ada hematocrit, idan akwai azãba mai tsanani itching, ko ya karu thrombocytosis.

Bargo dasawa da aka yi a wannan cuta ne musamman rare, tun da Pathology a cikin hali na isasshen magani ne ba na mutuwa.

Ya kamata a lura da musamman magani regimen a kowane hali zabi akayi daban-daban. A sama-aka bayyana far ne ga bayani dalilai kawai. An ba da shawarar a yi kokarin magance wannan cuta.

zai yiwu rikitarwa

Haka kuma cutar mai tsanani ne isa, don haka kada ka manta da magani. In ba haka ba, shi ne kara da alama na m rikitarwa. Wadannan sun hada da wadannan:

• Jini clots. Sanadin pathological aiwatar iya lurking a kara jini danko, kara yawan ja da maikacin jini, da kuma platelets.
• Urolithiasis da gout.
• Zub da jini, ko bayan qananan m hanyoyin. Kamar yadda mai mulkin, irin wannan matsala fuskanci bayan hakori hakar.

Outlook

Vakeza cuta - a rare cuta. Alamun cewa faruwa a farkon matakai na ta ci gaba, ya zama sa ga nan da nan jarrabawa da kuma bi-up far. A rashin isasshen magani, idan cutar da aka ba kamu a dace hanya, mutuwa ya auku. Babban dalilin mutuwar sau da yawa aiki jijiyoyin bugun gini da rikitarwa ko canji na cuta a cikin wani na kullum cutar sankarar bargo. Duk da haka, m magani da kuma tsananin riko ga dukkan shawarwari daga likita zai iya muhimmanci mika rayuwar da haƙuri (15-20 shekaru).

Muna fatan cewa duk bayanin da aka bayar a cikin labarin zai zama gaske amfani a gare ku. Zauna lafiya!