Dystrophy - shi ne cewa a cikin magani? Hanta, zuciya, cornea, tsoka

Dystrophy - wata cuta a cikin abin da jikin kyallen takarda ya faru a cikin hasara ko jari na abubuwa da ba su da takamaiman ga al'ada jihar.

Lalacewar da Kwayoyin da intercellular abu take kaiwa zuwa gaskiya cewa waxannan cututtukan zuciya da sauransu daina aiki daidai. Dalili da wannan cuta ne trophic cuta (jerin matakai da cewa samar da muhimmanci ayyuka na Kwayoyin).

Sanadin

Wannan Pathology yana da yawa daban-daban haddasawa. Yana iya zama hereditary rayuwa cuta, cututtuka, danniya da hankulansu matsaloli, saukar da rigakafi, matsalar aiki na samfur na gastrointestinal fili, matalauta rage cin abinci, da mummunan tasiri na daban-daban dalilai, chromosomal da somatic cututtuka.

Ba daidai ba ne cewa dystrophy - a cutar da ke shafar kawai bai kai ba jariran. A ci gaba da irin wannan cuta na iya haifar da shafe tsawon azumi ko wuce haddi na carbohydrate-dauke da abinci a cikin ganyayyaki. A sabili da nakasar dystrophy iya zama ma matasa ko, a akasin haka, tsofaffi mahaifiyarsa.

cututtuka

Dangane da irin da kuma mai tsanani da cuta yana da uku digiri, wanda aka halin da wasu cututtuka.

A farko mataki - da mãsu haƙuri ne da rage a jiki nauyi da kuma tsoka sautin, kyallen takarda zama kasa na roba, gaji da damuwa da rigakafi da tsarin, za'a iya matsaloli tare da kujera.

Na biyu digiri - a kan bango na m nauyi asara zama sirara, mai yiwuwa ma ya bace subcutaneous nama, jiki ne yake shan wahala daga rashin bitamin.

Uku Degree - da kwayoyin da aka gaba daya tsautsayi, da mutum gaji da damuwa da numfashi da kuma zuciya aiki, rage jiki zafin jiki da kuma jini.

Bugu da kari, duk siffofin dystrophy suna tare da general cututtuka: da mãsu haƙuri kuma m, ya rage ko ba ci, gaji da damuwa da barci, akwai wani janar wani rauni, akwai canje-canje a cikin jiki nauyi, gastrointestinal cuta, rage rigakafi. Wannan mutum bai sani ba cewa ya dystrophy cututtuka zare kudi gajiya ko danniya.

rarrabuwa

Rarraba da cutar bisa ga dama sharudda. Saboda haka, bisa ga asalin Pathology ne nakasar da kuma samu, a kan ruwan dare daga cikin tsari - gida ko general, bisa ga musayar keta damu rarrabe gina jiki, mai, carbohydrate dystrophy. Ya danganta da inda aiwatar da aka sarrafa, cutar na iya zama salula (parenchymal), extracellular (mesenchymal, stromal-jijiyoyin bugun gini) da kuma gauraye.

Bugu da ƙari kuma, ya zama ruwan dare jinsunan kamar ciwon tamowa, kuma gipostatura paratrofiya.

Tamowa - ya fi na kowa nau'i na cutar. Shi ne m da samu. Halin da rashin jiki nauyi dangane da ci gaban da mãsu haƙuri.

Paratrofii lokacin da akwai wani wuce haddi na jiki nauyi, wadda aka sa ta tamowa, kuma metabolism.

Gipostatura bayyana a cikin wannan rashin nauyi da kuma tsawo daidai da sharudda a wasu shekaru.

nakasar dystrophy

Wannan yanayin ne sakamakon gaji cuta da metabolism sunadarai, fats ko carbohydrates. Lokacin da yaro ta jiki ba ya samun wani enzyme, jari na faruwa ba gaba daya narkewar kayayyakin da metabolism a kyallen takarda ko gabobin. Ko da kuwa da sarrafawa tsari, akwai ko da yaushe lalacewar da tsakiya m tsarin nama, wadda take kaiwa zuwa mutuwa.

Ya danganta da wurin da wadannan iri wannan cuta:

Cell (parenchymal) m degeneration

Wannan wata matsala ce da ya auku a lokacin wani rukuni na sel da suke da alhakin aiki na jiki, akwai wani na rayuwa cuta. Daga cikin abubuwan da tsokana da ci gaban cutar za a iya lura hypoxia, kamuwa da cuta, abin mãye (musamman barasa), matalauta abinci mai gina jiki da kuma bitamin rashi.

Daya misali na irin wannan Pathology ne degeneration na hanta. Yana bayyana kanta a cikin rashin iyawa na hanta Kwayoyin domin su aiwatar da ayyuka, abu don maye su m nama. Wannan zai iya sa kumburi da kuma cirrhosis.

M effects da m kuma mai guba nau'i na cutar. Jiha ta farko da ke da saurin ci gaban, sakamakon zai iya zama hanta gazawar. Mai guba hanta dystrophy ne cikin hanyar necrosis na ta Kwayoyin.

Dystrophy zuciya - wani misali daga cikin salon salula m degeneration. A wannan yanayin da tsokar lalacewa, ta yi aiki ne sosai. Zuciya ƙaruwa a size, da myocardium zama flabby, yellow, tashi daga endocardium tube saboda m shaida na mai. M degeneration na zuciya ne sakamakon hypoxia (oxygen rashi), cututtuka da kai ga rayuwa cuta, tamowa.

Protein parenchymal degeneration

An kasu kashi da dama iri:

- hyaline droplet degeneration halin da jari a cikin kodan (wani lokacin shan wahala daga hanta ko zuciya) furotin droplets. Wannan na faruwa a lokacin da koda cututtuka irin mahara myeloma, glomerulonephritis, amyloidosis. Cutar da yake boye da take kaiwa zuwa komowar sakamakon.

- Lokacin da hydropic (hydropic) dystrophy a gabobin (hanta, adrenal, myocardium, fata Kwayoyin) ya auku tari gina jiki ruwa droplets. Tare da wani wuce haddi daga gare su a cikin wani cell taso balan-balan degeneration (kwaya aka tilasta periphery). Sa a kama yanayin Can take hakki na ruwa-gishiri metabolism, hypoproteinemia, edema, cututtuka da kuma intoxications.

- Rogovaya dystrophy halin da samuwar da jaraba abu, inda shi ba zai iya zama, ko wuce kima jari na inda ya kamata ya kasance a cikin al'ada adadin (fata, kusoshi). Tsokane wata cuta iya beriberi, fata lahani na ci gaba, kwayar cututtuka, da kuma na kullum kumburi.

Nail dystrophy iya zama sakamakon kamuwa da cutar, fata cututtuka, malfunctions gastrointestinal, endocrine ko tsakiya m tsarin. Bugu da kari, wannan Pathology ne, wani lokacin tare da wasu cututtuka na zuciya da jijiyoyin jini da kuma Sistem tsarin.

Nail dystrophy iya zama nakasar ko samu. A nakasar nau'i na cutar na iya zama mai cikakken rashi na, thinning kuma thickening da ƙusa farantin. An samo cuta halin da hankali detachment da ƙusa farantin, ƙusa sako ko whitening. Bugu da ƙari, da ƙusa farantin iya zama ma convex, tare da mai gangara kuma a tsaye GROOVES. A dalilan da irin wannan cuta na iya zama eczema, psoriasis, lichen planus, ƙusa rauni.

Cornea degeneration yana da dama iri:

- Hyperkeratosis - wani wuce haddi na keratin a kan fata.

- nakasar hyperkeratosis na fata, da ake kira ichthyosis. jariri ta jiki da aka rufe a lokacin farin ciki fata cewa yayi kama kifi ma'auni.

- leukoplakia - wata cuta a cikin abin da mucosal coarsen. Wannan fallasa da cervix, esophagus, baki, da sauransu. D.

Parenchymal carbohydrate dystrophy

Yana da sakamakon musayar glycogen da glycoproteins take hakki. Take hakkin glycogen metabolism halayyar pathologies kamar ciwon sukari ko glycogenosis.

Saboda damuwa metabolism na glycoproteins a Kwayoyin sa jari mucoids da mucins (gamsai abu). Wannan ya faru a cututtuka irin rhinitis, gastritis, mashako, kuma haka a. D.

Extracellular (mesenchymal) dystrophy

Yana tasowa a cikin stroma (tushen kunshi connective nama) jikinsu. A wannan yanayin, da tsari ya shafi dukan nama da jini. An misali iya zama retinal dystrophy. A cuta na iya zama ko dai nakasar ko samu. A cikin mutane, rage gani acuity, yana da wuya a kewaya a cikin duhu. A karshe mataki da cuta ne retinal detachment idanu ko pigmentalnaya dystrophy. Sau da yawa da hannu a cikin tsari da kuma cornea.

corneal dystrophy halin da shirin hana haske, rage gani acuity da kuma ji na ƙwarai. Yana iya zama hereditary ko samu. Cutar progresses hanzari, kamawa biyu idanu, don haka yana da muhimmanci sosai dace ganewar asali.

Alamun, wanda ya kula - wannan shi ne zafi da kasashen waje jiki abin mamaki a cikin idanu, da jan launi da idanu, na ruwa idanu, tsoro na haske, ta rage hangen nesa.

Secondary corneal dystrophy taso a matsayin sakamako na tagayyara. Yana iya kuma zama saboda bitamin rashi, canjawa wuri collagen cututtuka, nakasar glaucoma.

Murdede dystrophy - a yanayin da cewa yana da halin m tsoka rauni da degeneration. Mafi na kowa iri biyu.

Duchenne murdede dystrophy

Yafi auku a yara. A wannan cuta akwai wata karuwa maraƙi tsokoki saboda mai tarawa da kuma yaduwa na connective nama. A cuta ne sosai tsanani, kuma ya ci gaba cikin sauri, shekaru 12 a cikin haƙuri hasarar da ikon matsar kusa. 'Yan mutane rayu tsawon fiye da shekaru ashirin.

Steinert ta cuta

Shi ne ya fi kowa a cikin manya. A cutar za a iya gaji, ta bayyana a kowane zamani. Daga cikin na farko da bayyanar cututtuka iya lura myotonia (bayan yankan tsokoki shakata sosai sannu a hankali) da kuma wani rauni da fuska tsokoki. Progressive degeneration na tsokoki sannu a hankali, rinjayar da m tsokoki da kuma zuciya tsoka.

Lokacin da mesenchymal gina jiki dystrophy iya sha hanta, koda, saifa, kuma adrenal gland mutum. Mazan mutane sun lalace cikin zuciya da kwakwalwa.

Stromal jijiyoyin bugun gini m degeneration - wannan abin da ya faru mai raɗaɗi nodular adibas a kan mutum wata gabar jiki (msl, dercum cuta). Cutar na iya zama na gida da kuma general, don su tsokane jari na abubuwa, kuma su hasãra.

A sakamakon mesenchymal carbohydrate dystrophy iya zama edema, kumburi ko softening daga cikin gidajen abinci, ƙasũsuwa da, guringuntsi mutum.

Magani da kuma rigakafin

Bayan ka saita daidai ganewar asali, shi wajibi ne a ci gaba nan da nan don lura da wannan cuta dangane da irin da kuma mai tsanani. Zabi da dabara na magani zai taimaka wani gwani, amma ga wani nau'i na dystrophy bukatar tsara kula dace ga marasa lafiya, don kawar da duk abubuwan da abin da zai iya tsokana rikitarwa.

Bugu da kari a gudanar da asali magani ne ma wajibi ne don tsayar da tsarin mulki na rana, da kuma tsaya wa a tsananin rage cin abinci, dangana ga likita, da ake bukata sabo ne iska, hydrotherapy, motsa jiki.

Ga masu rigakafin wannan cuta ga nasu kiwon lafiya ya kamata a hankali da sanya idanu, har zuwa yiwu don kawar da duk korau abubuwan da za su iya fararwa degeneration na kowane irin.

Qarfafa rigakafi da tsarin, ci wani m da kuma daidaita, motsa jiki da kuma guje wa danniya - wannan zai zama mafi kyau rigakafin ne ba kawai wannan ba, amma kuma da yawa wasu cututtuka.