Fat embolism, thromboembolism

Embolism kira m jijiyoyin bugun gini occlusion, sakamakon lalace jini ya kwarara zuwa nama ko zuciya da sauransu. Fat embolism, bi da bi, wani blockage na jini mai. Yadda aka saba, wannan mai kai-asali, shi ne musamman rare - waje (artificially gabatar a cikin jikinsu kamar yadda sauran ƙarfi, ko da kwayoyi don bincike dalilai).

Fat embolism ne sakamakon traumatic rikitarwa. Yawancin lokaci shi ake dangantawa da tsanani kwarangwal rauni (samu karaya na hip, kafafuwa, tibia, crushing kitse). Na iya faruwa a lokacin da pancreatitis, ciwon sukari, septicemia, bugu jihohi, anemia, postresuscitative lokaci, sakamakon wani erroneous gwamnatin sia-narkewa kwayoyi, liposuction.

Fat embolism kamu kasa akai-akai fiye da ya auku saboda "masked" kuma karkashin wasu yanayi. Alal misali, a karkashin ciwon huhu.

A bambanci: a walƙiya siffar (sakamakon mutuwa a cikin 'yan mintuna). m siffar (ci gaba bayan rauni a karo na farko). subacute form (latent lokaci na 12-72 hours.)

Asibiti kasaftawa na huhu, cerebral da kuma gauraye (fi na kowa) form.

Fat embolism, magani

Jiyya ne da nufin farko a goyon bayan da harshen damo. Wannan wucin gadi huhu iska, dubagari na trachea da bronchi. Gabatar don magance hyperpyrexia lytic cakuda. Capillary permeability a high allurai rage steroid hormones inganta disaggregation jini Kwayoyin. Infusions na albumin, reopoliglyukina, glucose mafita yi sosai a hankali saboda da blockage na jini da kuma mai barazana obalodi. Input yana nufin wanda normalize jini rheology (aminophylline, pentoxifylline, nicotinic acid, xantinol nicotinate, glyukozonovokainovaya cakuda). Heparin kamata ba a yi amfani da, shi ne worsen huhu aiki da kuma kara da matakin na m acid. Lipostabil stabilized kitse, alli chloride, deholin. Idan sami kitse embolism, dole ne ka kauce daga aiki hanyoyi na karaya kam, tabbatar da immobilization ta wasu hanyoyin. Karfafawa na samu karaya, da za'ayi a cikin farkon matakai, za su yi gargaɗi da kitsen embolism da kuma kawar da hypoxia.

Thromboembolism, cututtuka da kuma haddasawa.

Mafi sau da yawa bayyana da hankula cututtuka: shortness na numfashi, kwatsam shortness na numfashi ya auku. A supine matsayi da haƙuri ji dan sauki. Zai yiwu ciwon kirji (daban-daban hali - daga m? Unci ga ciwo mai tsanani). A rare lokuta kuma zai iya zama hemoptysis. Yiwu wani buɗi na cyanosis - hanci, lebe, kunnuwa zama kodadde ko bluish launi. Akwai tari, m bugun zuciya rãyar, da mãsu haƙuri jefa cikin wani sanyi gumi, ya ragae saukar, m, drowsy. Iya samun cramps a kafafu da kuma asarar sani.

A sabili da thromboembolic events ne jini clots cewa faruwa a jijiyoyinmu. A thrombus za a iya kafa a sakamakon lalacewar jijiyoyin bugun gini ganuwar, jini ya kwarara da aka ragae saukar a wannan wuri da kuma akwai gudan jini (jini clots). Ganuwar da jijiyoyinmu iya lalace a iri daban-daban kumburi cututtuka, raunin da ya faru, injections. Slow kwarara za a iya lalacewa ta hanyar zuciya rashin cin nasara, da shafe tsawon matsayi (a zaune, kwance). Karuwan jini clotting iya zama hereditary da kuma rashin cin nasara a cikin jini coagulation tsarin.

Hadarin dalilai: m shekaru, malignancy, tiyata, ciki, rauni, cuta (lupus, erythremia, nephrotic ciwo, hemoglobinuria).

A matakin na halaka ne m da ECG, X-ray, samun iska-perfusion huhu scintigraphy. Jiyya ne da za'ayi kawai a wani asibiti. Abin baƙin ciki, har ma da dace fitarwa ba ko da yaushe samar da wani cin nasara magani. Don kauce wa mutuwa kana bukatar ka nemi taimako maza maza. Kamar yadda gaggawa gabatar for 10 000 raka'a heparin intravenously, arin ake bukata.

A labarin da aka rubuta domin reference kawai. Don Allah ya bayyana bayyanar cututtuka kanemi shawara. Kai-bincike da kuma kai-magani zai iya kai wa ga ban tausayi sakamakon!