M hagu na ramin zuciya gazawar

The zuciya tsoka, aka sani ya yi wani irin famfo aiki a jikin mu. Idan na'urar na duk abin da dalili, ba zai iya jimre da su nan da nan aiki, dole mu yi zuciya rashin cin nasara. A kowace shekara wannan Pathology kawai ƙaramin, kuma mafi sau da yawa shi take kaiwa zuwa kwanan nan mutuwa.

M zuciya rashin cin nasara. Mene ne wannan?

M zuciya rashin cin nasara - da shi sun bayyana ne kwatsam wani karu da contractile aiki na zuciya, sa ta obalodi da na huhu wurare dabam dabam cuta. Cutar na iya bayyana a matsayin a kan bango na data kasance cardiac munanan, kuma babu gabata cardiac ailments. Mafi sau da yawa shi ne kamu da tsokar zuciya infarction faruwa a lokacin da cell mutuwa saboda m tabarbarewar jini wurare dabam dabam a cikin wannan yanki.

Dangane da obalodi sassan babban tsokoki na jikin mutum, akwai biyu main hanya da cuta: dama na ramin zuciya da kuma bar na ramin zuciya gazawar. A karshen, mu tattauna a zurfafe a cikin wannan labarin.

A pathogenesis da cutar

Daya daga cikin manyan pathogenetic al'amurran bar na ramin zuciya gazawar da karuwa a hydrostatic matsa lamba a cikin capillary da venous sassa na huhu wurare dabam dabam. A na huhu interstitium hankali tara ruwa a wuce haddi, wadda take kaiwa zuwa runtse jikin elasticity. Huhu zama mafi "wuya", akwai hana cuta. Sa'an nan edematous m shiga cikin Alveoli, sabõda haka suka daina shiga a gas musayar.

Shi ne m yaduwa tare serous huhu da Bronchial kututturan. Akwai infiltration na serous ruwa da kuma perivascular peribronchial sarari, wanda aka nuna a cikin su kai tsaye juriya tabarbarewar musayar yanayi.

A jini daga hagu atrium unventilated Alveoli gauraye da oxygen-arziki abu. Wannan entails a rage a cikin jimlar jijiya oxygen tashin hankali, hypoxemia ci gaba. A can farko yana da Sistem karshe hali.

Hagu na ramin zuciya kasawa mafi yawa ana sa da tsanani overloads ko dacewa sassa na zuciya raunuka. Irin wannan cuta entails matalauta wurare dabam dabam. Yana bukatar ba kawai dace ganewar asali, amma kuma m magani. Lokacin hade tare da cuta-gefe zuciya rashin cin nasara qara yiwuwar mutuwa daga mãsu haƙuri.

Babban Sanadin cutar

• Cardiomyopathy.
• CHD.
• Tsokar zuciya infarction.
• Cardiogenic na huhu edema.
• Symptomatic jijiya hauhawar jini.
• Mitral stenosis.
• Major tiyata.
• kwakwalwa rauni.
• Kamuwa da yanayin da cutar.
• Mai guba effects a kan myocardium (magani, barasa amfani).

M hagu na ramin zuciya gazawar da kuma tsokar zuciya infarction

Haka kuma cutar sau da yawa tasowa a kan bango na tsokar zuciya infarction. Yawancin lokaci shi ne saboda wani illa diastolic aiki da kuma iya ci gaba da wata al'ada ejection sulusi da murabba'i. Muhimmiyar rawa a ci gaban da zuciya rashin cin nasara a wannan harka da aka ba mitral regurgitation. Shi ne saboda ischemia na papillary tsokoki ko asarar gagarumin tsokar taro.

A cuta ne a cikin farkon hours a marasa lafiya tare da tsokar zuciya infarction ne halin da kaifi karu a zagawa da jini girma, canje-canje a cardiac fitarwa da kuma wani m sodium riƙewa.

asibiti hoto

An sani cewa kusan duk cututtuka da na kowa hali na cardiac cututtuka, watau zafi da aka bayyana a cikin zuciya. Yana iya ba a hannu, wuyansa, ko kafada. Wannan sabon abu ne yawanci lura a wannan cuta kamar hagu na ramin zuciya gazawar. Alamun suna complemented zazzabi, shortness na numfashi, kwatsam matsa lamba surges da kuma yawan zafin jiki take hakkin.

A yanayi da tsanani da cuta cututtuka kai tsaye dogara ne a kan daidai da abin da rabo daga zuciya tsoka ta lalace. Domin hagu na ramin zuciya siffar, ban da babban bayyanar cututtuka, halin da bayyanar syncope, arrhythmia, juwa ko jiri, kumburi na extremities, ta ƙara lakar jijiyoyinmu.

A hankali, duk da ãyõyin hagu na ramin zuciya kasawa aka inganta, bayyana yanzu, ba kawai a mataki na aiki, amma kuma a sauran. Idan ba ka amsa ga wannan matsalar a dace hanya, ba za a iya mulki mutuwa.

siffofin da cutar

A Pathology iya faruwa a dama iri.

• Tare da m irin hemodynamics. Yana bayyana a cikin nau'i na cardiac fuka (fuka-haren), da kuma na huhu edema.
• Tare da hypokinetic hemodynamics:

1. Arrhythmic buga. Yana tasowa a mayar da martani ga take hakkin zuciya rate.
2. Reflex buga. Yana bayyana a matsayin mayar da martani ga zafi.
3. Gaskiya cardiogenic buga. Faruwa a lokacin gagarumin yawa na lalacewa (a kan 50% hagu na ramin zuciya taro).

• A kaifi tabarbarewar a riga kamu da zuciya rashin cin nasara (tsoka ba ya samar da sashen tsarin yana bukatar isasshen jini wadata).

bincikowa da

Mutane da yawa cardiac Pathology yanayi ne daban-daban m ci gaban, kuma m hagu na ramin zuciya kasawa ba wani togiya a wannan batun. Doctor idan ka zargin wannan ganewar asali ne abu na farko da yake don su bincika haƙuri. Duk da haka, da jiki da jarrabawa ne ba su isa ba a tantance da cikakken asibiti hoto. Saboda haka, da mãsu haƙuri An wajabta a yawan bincike hanyoyin, ciki har da ECG, zuciya duban dan tayi, x-ray na sternum, kuma radionuclide ventriculography so.

likita zai iya tabbatar da bincike da kuma m jiyya a kan tushen da sakamakon samu.

M hagu na ramin zuciya kasawa: gaggawa kula

A lokacin harin da haƙuri da muhimmanci sosai lokaci don samar da gwani taimako ga tawagar likitoci da ewa ba. A farko wuri shi ya kamata a sa a kan gado (na ƙarya tsananin haramta), samar da kwarara oxygen far da su fara.

A haƙuri ya kamata a ba "Nitroglycerin" a karkashin harshe, shigar subcutaneously 1% "Morphine". Tare da ci gaba mai tsanani na huhu edema saboda da rage a cikin jini yana gabatar da "prednisolone" intravenously (5.3 ml).

A babu muhimman magunguna da haƙuri kamata amfani da wani tourniquet da kwatangwalo, kuma kafadu zuwa sharply iyakance adadin zagawa da jini. Har ila yau intravenously iya shigar da musamman saje. A saboda wannan dalili, a 10 ml sirinji dole gwada 3 ml na 96% ethanol, da kuma bayan da ƙara ruwa ga allura. Yana da muhimmanci cewa dukan girma da aka cika. Wannan bayani yana mai defoaming sakamako. A haƙuri ya zama dalili cardiac likita birged. Bayan kawar da na huhu edema , dukan marasa lafiya da ganewar asali "m hagu na ramin zuciya zuciya kasawa" ne batun m asibiti. Transport mafi yawa ana da za'ayi a kan wani gadon ɗauka maras lafiya a zaune matsayi.

Ka'idodi na magani

Wannan Pathology barazanar haƙuri da rayuwa da kuma ko da yaushe bukatar gaggawa magani.

• Idan hanyar da cuta ne wani canji a cikin zuciya rate dattako hemodynamics da kuma yanayin haƙuri wajibi ne a mayar da zuciya rate.
• Idan dalili ne ya faru kafin tsokar zuciya infarction, domin magance cutar wajibi ne don normalize jijiyoyin zuciya jini ya kwarara da sauri. A prehospital kula da yanayi yana yiwuwa ta hanyar tsari thrombolysis, Ina nufin igiyar jini shirye na musamman ta soke jini clots.
• Idan bar na ramin zuciya kasawa ne sakamakon da baya samo asali cuta intracardiac hemodynamics saboda discontinuities infarction ko rauni da aka nuna nan da nan asibiti.

miyagun ƙwayoyi far

Jiyya da cutar, in general, ya shafi wani Hadakar m (cikakken sauran, babu tsanani jiki lodi, girmama likita rage cin abinci). Duk da haka, shi ne mafi m matakan, wanda wani lokacin ba ma iya tsara da zafi cewa accompanies da cuta daga m hagu na ramin zuciya gazawar. Jiyya tare da kwayoyi kawai a cikin wannan hali, ya je ceto.

Wadannan su ne main manufofin kiwon lafiya far:

1. Daidaita tsokar contractility.
2. Kawar da bayyanar cututtuka na sinus tachycardia.
3. Rigakafin thrombus samuwar kuma jijiyoyin bugun gini occlusion.
4. Daidaita aiwatar ruwa tukar tumbi daga jiki.
5. Renewals gafarta musu.

Da farko, ba tare da togiya, dukan marasa lafiya wajabta oxygen far. Don magance haddi ruwa a jiki bayar da shawarar shan diuretics ( "Furosemide", "Uregei") da kuma mai tsanani ƙuntata m ci. Don rage jijiyoyin bugun gini juriya da kuma a lokaci guda don inganta jini wurare dabam dabam, rubũta vasodilators da Antiplatelet jamiái ( "tiklid", "Plavix"). Don bunkasa tsokar contractility wajabta cardiac glycosides da cardiotonics.

All kwayoyi likita ta zayyana wani mutum akai ga kowane haƙuri. Saboda haka, yana da muhimmanci a fahimci cewa surface kai-magani iya kawai worsen da na asibiti hoto da kuma samar da ake so sakamakon.

Lokacin da ka bukatar tiyata?

Idan magani ba taimaka haƙuri da yanayin, bar na ramin zuciya zuciya gazawar kawai ƙarfafa a cikin manifestations, bayar da shawarar tiyata. A halin yanzu a asibiti yi ne biyu daban-daban na yadda ake gudanar: cardiomyoplasty da kafawa na musamman na'urar inganta jini wurare dabam dabam. Dukansu zažužžukan su ne rai barazana, don haka ne kawai cikin matsanancin koma ga su taimako. Bugu da ƙari, post-gudanar da sakamako m kawai bayan 3-4 watanni bayan wani dogon dawo da lokaci. Kafin mu je a kan irin wannan aiki, wajibi ne don tuntubar likita da kuma kimanta duk da abũbuwan amfãni, kuma zai yiwu disadvantages.

rikitarwa

Idan wani m kamu da "hagu na ramin zuciya rashin cin nasara," ya sakaci nasa kiwon lafiya da kuma magani jinkiri, m farkon mutuwa.

m matakan

A gaban cututtukan zuciya da na kullum yanayin da tsarin da aka nuna a kai a kai da sanya idanu a likitan zuciyar, da kuma tsananin riko ga duk da kayan abinci. Shi ne ya zama wajibi domin rage da tasiri a kan dukan jiki. Wadannan sun hada da addictions (shan taba, shan barasa da kwayoyi), Psycho-wani tunanin obalodi da m danniya da kuma kiba.

Kamar yadda prophylaxis na irin cututtuka a matsayin m hagu na ramin zuciya rashin cin nasara, wanda ya haddasa sukan boye a hypodynamia sashi shawarar motsa jiki. Yana iya zama iyo, tafiya a waje, gudun kan, wasa a cikin dakin motsa jiki, ko yoga. Wajibi ne a kullum saka idanu jini. dole ne ka biya musamman da hankali ga abinci mai gina jiki. A rage cin abinci ya zama kamar kyau-daidaita, high-fiber abinci. Yana da kyau a daina m da soyayyen abinci, kazalika da overly pungent abinci.

Wadannan sauki m matakan ba zai iya kawai hana ci gaban irin wannan hadari cuta ga kiwon lafiya, amma kuma inganta] aukacin halin rayuwar.

ƙarshe

A cikin wannan labarin, za mu yi magana game da abin da yake akwai wata cuta da ake kira hagu na ramin zuciya gazawar. A Sanadin wannan cuta na iya zama sosai daban-daban, kama daga na tsokar zuciya infarction da kuma kawo karshen mai guba tasiri a kan kwayoyin kwayoyi. Dace ganewar asali da kuma m magani rage girman hadarin da rikitarwa, mafi hatsari wanda yake shi ne m. Zauna lafiya!